Amparo
Amparo Alfonso Rancaño, Llugo ES
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20110065138 | Quantitative Method For Detecting Yessotoxins In Fishery Products On Based On The Activation That The Toxin Produces In Cellular Phosphodiesterases And Therapeutic Use Of This Activation - The present invention relates to a quantitative method for detecting yessotoxins in fishery products based on the activation the toxin produces on cellular phosphodiesterases and the therapeutic use of this activation. The cellular target of yessotoxin (YTX) and its analogs is the activation of phosphodiesterases (PDEs). The PDEs-YTX bond produces a measurable signal. The bond can be quantified by means of an affinity biosensor or by fluorescence. The biosensor detects biomolecular interactions and allows determining the presence of YTX due to its interaction with PDEs. The variations in the degradation rate of the fluorescent derivative anthraniloyl-cAMP are determined by means of plate fluorescence. The rate at which the PDEs degrade this molecule increases in the presence of YTX. YTX inhibits immunological activation of mastocytes in rats and induces a cytotoxic effect in human hepatocarcinoma cells, which implies two therapeutic uses of YTXs as an antiallergic and antitumor compound. | 03-17-2011 |
Amparo Alfonso Rancaño, Llugo ES
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20110065138 | Quantitative Method For Detecting Yessotoxins In Fishery Products On Based On The Activation That The Toxin Produces In Cellular Phosphodiesterases And Therapeutic Use Of This Activation - The present invention relates to a quantitative method for detecting yessotoxins in fishery products based on the activation the toxin produces on cellular phosphodiesterases and the therapeutic use of this activation. The cellular target of yessotoxin (YTX) and its analogs is the activation of phosphodiesterases (PDEs). The PDEs-YTX bond produces a measurable signal. The bond can be quantified by means of an affinity biosensor or by fluorescence. The biosensor detects biomolecular interactions and allows determining the presence of YTX due to its interaction with PDEs. The variations in the degradation rate of the fluorescent derivative anthraniloyl-cAMP are determined by means of plate fluorescence. The rate at which the PDEs degrade this molecule increases in the presence of YTX. YTX inhibits immunological activation of mastocytes in rats and induces a cytotoxic effect in human hepatocarcinoma cells, which implies two therapeutic uses of YTXs as an antiallergic and antitumor compound. | 03-17-2011 |
Amparo Andrés Bello, Valencia ES
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20130196045 | EMULSIFIED SPREAD BASED ON OLIVE OIL AND/OR OTHER VEGETABLE OILS AND METHOD FOR PREPARING IT - The present invention belongs to the field of food spreads, more specifically it relates to an emulsified spread based on olive oil that is not subjected to hydrogenation or hydroxylation, as well as to a method for preparing it. Optionally, the olive oil of the composition can be partially or totally substituted with any other vegetable oil. The spread of the present invention does not require cold storage. | 08-01-2013 |
Amparo Andrés Bello, Valencia ES
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20130196045 | EMULSIFIED SPREAD BASED ON OLIVE OIL AND/OR OTHER VEGETABLE OILS AND METHOD FOR PREPARING IT - The present invention belongs to the field of food spreads, more specifically it relates to an emulsified spread based on olive oil that is not subjected to hydrogenation or hydroxylation, as well as to a method for preparing it. Optionally, the olive oil of the composition can be partially or totally substituted with any other vegetable oil. The spread of the present invention does not require cold storage. | 08-01-2013 |
Amparo Chiralt Boix, Valencia ES
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20130196045 | EMULSIFIED SPREAD BASED ON OLIVE OIL AND/OR OTHER VEGETABLE OILS AND METHOD FOR PREPARING IT - The present invention belongs to the field of food spreads, more specifically it relates to an emulsified spread based on olive oil that is not subjected to hydrogenation or hydroxylation, as well as to a method for preparing it. Optionally, the olive oil of the composition can be partially or totally substituted with any other vegetable oil. The spread of the present invention does not require cold storage. | 08-01-2013 |
Amparo Fuertes Miquel, Sant Cugat Del Valles ES
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20100148111 | Ce-N-O SYSTEM DERIVED FROM DOPING OF CERIUM OXIDE WITH NITROGEN HAVING GENERAL FORMULA CeO2-x-yNx - Ce-N-O system derived from doping of cerium oxide with nitrogen with general formula CeO | 06-17-2010 |
Amparo Gallardo-Moreno, Badajoz ES
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20110008801 | METHODS FOR MEASURING CELL-CELL OR CELL-MATRIX ADHESIVE FORCES AND COMPOUNDS FOR DISRUPTING ADHESIVE FORCES IN BIOLOGICAL SYSTEMS - The invention provides an atomic force microscope-based bioassay, assisted by thermodynamic characterizations to quickly and accurately screen for compounds that disrupt cell-cell or cell-substrate interactions. | 01-13-2011 |
Amparo Garcia López, Valencia ES
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20130143825 | COMPOUNDS FOR USE IN THE TREATMENT OF DISEASES BASED ON THE EXPRESSION OF TOXIC TRANSCRIPTS - Compounds to be used in the treatment of diseases based on the expression of toxic transcripts. The present invention relates to peptide molecules, specifically hexapeptides, designed for the prevention and/or treatment of diseases the etiology whereof is based on the presence of toxic transcripts that comprise CUG, CCUG, CGG, CAG and AAG repeats, preferably: DM1 SCA8, DM2, FXTAS, HD and FA. | 06-06-2013 |
Amparo Garcia López, Valencia ES
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20130143825 | COMPOUNDS FOR USE IN THE TREATMENT OF DISEASES BASED ON THE EXPRESSION OF TOXIC TRANSCRIPTS - Compounds to be used in the treatment of diseases based on the expression of toxic transcripts. The present invention relates to peptide molecules, specifically hexapeptides, designed for the prevention and/or treatment of diseases the etiology whereof is based on the presence of toxic transcripts that comprise CUG, CCUG, CGG, CAG and AAG repeats, preferably: DM1 SCA8, DM2, FXTAS, HD and FA. | 06-06-2013 |
Amparo GÓmez Siurana, Alicante ES
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20150320108 | ALUMINOSILICATE SAB-15 AS AN ADDITIVE FOR REDUCING THE TOXIC AND CARCINOGENIC COMPOUNDS PRESENT IN TOBACCO SMOKE - The invention relates to the use of the aluminosilicate SAB-15, or the acidic or sodic forms thereof, interchanged with Fe, Na, K, Ca, Ce, Zr, the oxides of Fe, Na, K, Ca, Ce, Zr, and mixtures of same as an additive for reducing the toxic and carcinogenic substances present in tobacco smoke. | 11-12-2015 |
Amparo Lopez Rubio, Paterna (valencia) ES
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20130085212 | PROCEDURE FOR THE OBTAINMENT OF NANOCOMPOSITE MATERIALS - The present invention relates to a procedure for the obtainment of a nanocomposite material through the technique of melt mixing comprising a polymeric matrix and a nanoreinforcement which has been previously dispersed in the same plastic or other matrix by means of electrospinning methods. | 04-04-2013 |
Amparo Pérez Díaz, Santiago De Compostela ES
Amparo Pérez Díaz, Santiago De Compostela ES
Amparo Pons Marti, Valencia ES
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20140347628 | MULTI-VIEW FUNDUS CAMERA - The invention relates to a fundus camera that incorporates an integral imaging system for capturing an integral photograph of the fundus. This integral photograph enables projecting a three-dimensional image and generating topographical maps of the fundus. The fundus camera can function plenoptically and thus zoom onto a two-dimensional image generated from the integral photograph. The proposed equipment consists of an optical system for illuminating the fundus and an optical system forming the integral image capture system. This capture system includes an ophthalmoscopic lens, a microlens array and a sensor, and it allows recording, in a single shot, multiple views of the fundus. To improve the resolution of the integral photograph, the capture system has a device that allows displacing the microlens array by a few microns. | 11-27-2014 |
Amparo Sanchez Navarro, Salamanca ES
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20090012326 | Use of Salicylate as an Antidote for Paraquat Intoxications in Mammals - The present invention concerns to the use of salicylate in the treatment of mammal intoxications caused by the herbicide paraquat (PQ). It was achieved, for the first time, 100% of survival 30 days after the administration to Wistar rats, by intraperitoneal route, of a PQ dose that, in the absence of treatment, is itself 100% lethal at the end of 6 days. The administration of salicylate, two hours after PQ, reversed the PQ toxicity and extended the life of the animals to the levels of the control group. | 01-08-2009 |